Stephen Juvet
Respirologist and clinician-scientist with the Toronto Lung Transplant Program who studies mechanisms of chronic rejection in transplanted lungs.
As of . Primary source: UHN Research profile.
Summary
According to his University of Toronto and UHN Research profiles, Stephen Juvet (MD, PhD, FRCPC) is a clinician-scientist and respirologist with the Toronto Lung Transplant Program and an Associate Professor at the University of Toronto. His stated research focus is mechanisms of chronic rejection and the determinants of immunological tolerance in transplanted lungs, using rodent models and a biobank of human specimens.
Details
- role
- Respirologist and clinician-scientist; Associate Professor (University of Toronto)
- affiliation
- Toronto Lung Transplant Program, University Health Network; University of Toronto
Sources and links
Related
Links from this record
- member of: Toronto Lung Transplant Program (University Health Network)
- author: Identification and characterization of inflammatory LILR and fibrotic SPP1 macrophages in chronic lung allograft dysfunction
- author: Emergence of a senescent and inflammatory pulmonary CD4+ T cell population prior to lung allograft failure
- author: Bronchoalveolar lavage cytokine-based risk stratification of clinically-stable lung transplant recipients with undefined rejection: Further insights from a follow-up investigation
- author: Detailed cellular and spatial characterization of chronic lung allograft dysfunction using imaging mass cytometry
- author: The CD8+ T cell content of transbronchial biopsies from patients with a first episode of clinically stable grade A1 cellular rejection is associated with future chronic lung allograft dysfunction
- author: Donor and recipient human leukocyte antigen-G polymorphisms modulate the risk of adverse immunologic events following lung transplantation
Linked from (derived)
- coauthor: The CD8+ T cell content of transbronchial biopsies from patients with a first episode of clinically stable grade A1 cellular rejection is associated with future chronic lung allograft dysfunction
- author: Identification and characterization of inflammatory LILR and fibrotic SPP1 macrophages in chronic lung allograft dysfunction
- author: Distinct fibrotic, epithelial and immune transcriptomic programs in phenotypes of chronic lung allograft dysfunction
- author (sole): Toward molecular endotypes of CLAD: dancing around the truth hidden in the tissue
- coauthor: Epithelial cell death markers in bronchoalveolar lavage correlate with chronic lung allograft dysfunction subtypes and survival in lung transplant recipients-a single-center retrospective cohort study
- coauthor: The impact of first untreated subclinical minimal acute rejection on risk for chronic lung allograft dysfunction or death after lung transplantation.
- coauthor: Bronchoalveolar lavage cytokine-based risk stratification of clinically-stable lung transplant recipients with undefined rejection: Further insights from a follow-up investigation
- coauthor: Spectrum of chronic lung allograft pathology in a mouse minor-mismatched orthotopic lung transplant model
- coauthor: Local intragraft humoral immune responses in chronic lung allograft dysfunction.
- coauthor: Interferon-stimulated and metallothionein-expressing macrophages are associated with acute and chronic allograft dysfunction after lung transplantation
- coauthor: Emergence of a senescent and inflammatory pulmonary CD4+ T cell population prior to lung allograft failure
- coauthor: Spectrum of chronic lung allograft dysfunction pathology in human lung transplantation.
- coauthor: Detailed cellular and spatial characterization of chronic lung allograft dysfunction using imaging mass cytometry
- coauthor: Donor and recipient human leukocyte antigen-G polymorphisms modulate the risk of adverse immunologic events following lung transplantation
- coauthor: Humoral immunity to lung antigens early post-transplant confers risk for chronic lung allograft dysfunction.
- member: Toronto Lung Transplant Program (University Health Network)
Known gaps in this record
- current funding
- ORCID
Information resource only. Not medical advice. Not a substitute for the care of the patient's transplant team.