Aspiration
Status: proposedEvidence level: proposed framework, abstract only
Aspiration is named in the review abstract among the pathways involved in CLAD. Proposed framework, abstract only.
As of . Primary source: Burman et al. 2026, J Heart Lung Transplant (DOI; abstract only was read).
Summary
The abstract of the review by Burman and colleagues names aspiration, the entry of stomach contents or secretions into the lungs, among the pathways involved in CLAD. The abstract does not state the strength of the evidence for this pathway, and the full text was not read. Only the abstract of the source review was read; the full text was not read. Status: proposed framework.
Sources and links
Related
Links from this record
- described in (abstract only): CLAD: translating basic science to clinical practice
- part of proposed framework: Molecular endotypes of CLAD (proposed framework)
Linked from (derived)
- pathway named in abstract: Molecular endotypes of CLAD (proposed framework)
- related to: Gastro-oesophageal reflux, oesophageal dysmotility and aspiration
- supports: Gastro-oesophageal reflux and gastric aspiration in lung transplant patients with or without chronic rejection
- describes: Triaging donor lungs based on a microaspiration signature that predicts adverse recipient outcome
- supports: Oesophageal stasis is a risk factor for chronic lung allograft dysfunction and allograft failure in lung transplant recipients.
- describes: Airway pepsinogen A4 identifies lung transplant recipients with microaspiration and predicts chronic lung allograft dysfunction
Known gaps in this record
- full text of the source review not read, so the evidence, citations and level of agreement for this item are missing
- funding and conflicts of interest of the source review not read
Information resource only. Not medical advice. Not a substitute for the care of the patient's transplant team.